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No IGF-1 / glucose impact: Unlike full GH, it does not appear to raise IGF-1 or impair glucose tolerance [5] [6]
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The relevant research question is whether intracellular NNMT inhibition produces pathway-specific effects under defined laboratory conditions, or whether broader changes in nicotinamide metabolism, NAD + availability, SAM-dependent methylation balance, and related biochemical pathways may influence additional cellular systems
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