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486 Worse still, the main challenge in the treatment of Alzheimers disease is that its pathogenesis has not been fully clarified, and the more recognized hypotheses of the pathogenesis include deposition of -amyloid (A) 487 and intracellular Tau protein aggregates, 488 neuroinflammation 489 and lack of acetylcholine (Ach), oxidative stress, 490 and bio-metal dyshomeostasis, etc

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760 These results together suggest that ACSL4 and LOX-catalyzed lipid peroxidation is involved in cerebral I/R injury, and inhibition of ACSL4 and LOX activity may be therapeutic targets for cerebral I/R injury

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