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Enzymes involved in lipid biosynthesissuch as CYP51A1are increasingly recognized as key modulators of cell survival and death, linking lipid homeostasis to the pathogenesis of diverse diseases (Fig
Each of these strategies can have a positive impact on cholesterol levels, but when combined, they can provide even greater benefits
We propose a unifying framework in which CYP51A1 integrates metabolic reprogramming and cell fate control, highlighting its potential as a therapeutic target across diverse human diseases
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