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Conversely, the resulting hyperglycemia and dyslipidemia further exacerbate OS through four key mechanisms: (1) the polyol pathway, where glucose conversion to sorbitol depletes NADPH and glutathione

Briefly, the pathogenesis of type 1 diabetes results from a triad of failures is a permissive genetic background in immunoregulation, a disruption of central and peripheral tolerance mechanisms, and an environmental insult that ignites and propagates a targeted immune attack on insulin-producing pancreatic beta cells

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