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Consequently, the therapy signifies the recovering effect BPC 157 has on occluded vessels (Vukojevic et al., 2018
The proposed mechanism involves controlling glutamate release in the synaptic cleft, modulating NMDA receptor stimulation, affecting excitatory amino acid transporter (EAAT)-mediated reuptake, and maintaining the glutamate/glutamine cycle within astrocytes
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(PubMed) Regulatory reports: the FDA (USA) highlighted multiple adverse events linked to compounded glutathione injections from mild to severe and life-threatening
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