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doi:10.1111/j.1478-3231.2006.01315.x
This article aims to elucidate the biochemical correlations between B complex deficiencies and neuropsychiatric manifestations ( Table 1 )
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257 Persistent YAP/TAZ activation is frequently observed in cancers such as HCC, breast cancer, and NSCLC
the excess free radicals deplete glutathione reserves, and the decreased ATP production slows glutathione replenishment, ultimately resulting in chronic glutathione deficiency [8]
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